Biblio

Found 7 results
Filters: Author is Chen, Shuhua and Keyword is Mice  [Clear All Filters]
2013
Ding F, Yao J, Rettberg JR, Chen S, Brinton RDiaz.  2013.  Early decline in glucose transport and metabolism precedes shift to ketogenic system in female aging and Alzheimer's mouse brain: implication for bioenergetic intervention.. PLoS One. 8(11):e79977.
Zhao L, Mao Z, Chen S, Schneider LS, Brinton RD.  2013.  Early intervention with an estrogen receptor β-selective phytoestrogenic formulation prolongs survival, improves spatial recognition memory, and slows progression of amyloid pathology in a female mouse model of Alzheimer's disease.. J Alzheimers Dis. 37(2):403-19.
Ding F, Yao J, Zhao L, Mao Z, Chen S, Brinton RDiaz.  2013.  Ovariectomy induces a shift in fuel availability and metabolism in the hippocampus of the female transgenic model of familial Alzheimer's.. PLoS One. 8(3):e59825.
Yao J, Zhao L, Mao Z, Chen S, Wong KCarmen, To J, Brinton RDiaz.  2013.  Potentiation of brain mitochondrial function by S-equol and R/S-equol estrogen receptor β-selective phytoSERM treatments.. Brain Res. 1514:128-41.
2012
Yao J, Irwin R, Chen S, Hamilton R, Cadenas E, Brinton RDiaz.  2012.  Ovarian hormone loss induces bioenergetic deficits and mitochondrial β-amyloid.. Neurobiol Aging. 33(8):1507-21.
2011
Zhao L, Yao J, Mao Z, Chen S, Wang Y, Brinton RDiaz.  2011.  17β-Estradiol regulates insulin-degrading enzyme expression via an ERβ/PI3-K pathway in hippocampus: relevance to Alzheimer's prevention.. Neurobiol Aging. 32(11):1949-63.
Yao J, Chen S, Mao Z, Cadenas E, Brinton RDiaz.  2011.  2-Deoxy-D-glucose treatment induces ketogenesis, sustains mitochondrial function, and reduces pathology in female mouse model of Alzheimer's disease.. PLoS One. 6(7):e21788.